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27 , moreover, they noted in these conditions reduced activity of succinate dehydrogenase (an enzyme particularly susceptible to oxidative damage) and an increased level of the oxidized form of glutathione (a cellular component of redox buffer)

In a clinical trial among COPD patients, significant improvement in forced expiratory volume (FEV1) occurred 5 minutes after dosing and continued to be evident through week 26 of treatment (D'Urzo 2011)